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8,9 Generally, released GLP-1 from intestinal L cells bind onto canonical receptors in the pancreatic islet, after oral glucose load, or may indirectly signal the hepatic vagal branch within intraportal vein, thereby promoting glucose-dependent insulin secretion from the pancreatic -cells, concurrently causing inhibition of glucagon secretion, and enhancing insulin gene transcription

ATGL is highly expressed in adipose tissues in contrast to non-adipose-derived tissues

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Sriharsha M, Reddy KN, Darsini TP, Reddy KS

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Condiciones como resistencia severa a la insulina, hipotiroidismo no controlado o uso de medicamentos que promueven ganancia de peso pueden contrarrestar los efectos del GLP-1